Atrial flutter and atypical atrial flutter
Gerry Kaye, Robert Lemery · 2018
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About 8,721 papers for “Atrial flutter”
Gerry Kaye, Robert Lemery · 2018
Cited by 0
David H. Spodick · The American Journal of Geriatric Cardiology · 2000
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Vance J. Plumb · Cardiac Electrophysiology Review · 1999
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Albert L. Waldo · Journal of Cardiovascular Electrophysiology · 1997
Entrainment was first described based on observations during rapid (overdrive) pacing of type I atrial flutter. Entrainment is capture of the reentrant circuit of a tachycardia without interrupting the tachycardia, so that with cessation of pacing, the spontaneous reentrant tachycardia is still present. During entrainment, the orthodromic wavefront from the pacing impulse resets the tachycardia to the pacing rate, while the antidromic wavefront either collides with the orthodromic wavefront of the previous beat (usual case) or is blocked by some other mechanism (refractoriness or another cause of block). Entrainment may be either manifest or concealed. The principles of entrainment during type I atrial flutter have permitted identification of targets for successful ablation, of mapping sites within or outside the reentrant circuit, and of appropriate pacing rates to successfully interrupt atrial flutter and restore sinus rhythm.
Cited by 96
Peter J. Papadakos, Mark L. Gestring · 2015
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2021
Atrial flutter is characterized on surface electrocardiogram (EKG) by often irregular but rapid supraventricular QRS complexes with a “sawtooth” baseline that represent flutter waves. Rapid atrial rates generally induce physiologic atrioventricular block (AVB). There are two types of atrial flutter based on the atrial rate. These include: type I AFL, and type II AFL. Due to the rapid atrial rate during AFL, functional/physiologic AVB is the rule and may result in a regular or irregular ventricular rhythm. AVB can change from a low to a higher grade or from a high to a lower grade of block. Slow supraventricular rhythm during AFL suggests third degree AVB with junctional escape rhythm. Atrial flutter may be mimicked by a variety of EKG artifacts. Baseline artifact from muscle tremors or panting may give the appearance of atrial flutter.
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Mithilesh K. Das, Douglas P. Zipes · Elsevier eBooks · 2012
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Erika Baiocco, Azzurra Fabbrizioli · Clinical cases in cardiology · 2015
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Ayman A. Hussein, Oussama M. Wazni · 2020
The incidence of atrial flutter (AFL) rises exponentially in relationship to advancing age but as it is for AF, is greatest with structural heart disease such as left atrial enlargement or ventricular dysfunction. This chapter discusses epidemiology and risk factors, clinical presentation, and diagnosis of AFL. Acutely, patients often complain of shortness of breath, palpitations, diaphoresis, chest discomfort, dizziness, and weakness. Patients may also complain of polyuria, which occurs as a result of increased atrial pressure from rapidly contracting atria against a closed AV valve, and the subsequent release of atrial natriuretic factor. The diagnosis of AFL is generally made on a 12-lead surface ECG by identifying flutter waves in leads II, III, aVF, and V1. When it is difficult to distinguish the flutter waves, slowing the ventricular rate, by using AV nodal blockers (e.g., adenosine or diltiazem) or vagal maneuvers (Valsalva or gentle carotid sinus message), may allow visualization of the flutter waves.
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Hubert Scharnagl, Winfried März, Markus Böhm et al. · 2009
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Jonathan S. Olshaker · Journal of Emergency Medicine · 1988
Cited by 2
R. Wayne Campbell · PubMed · 1998
Atrial flutter is a specific electrical entity. It has a now well-established mechanism involving a single macro-re-entrant circuit. Whilst many patients with atrial flutter may develop atrial fibrillation, atrial flutter deserves its own recognition and requires specific management. Acute termination by drugs is possible, although adding pacing improves efficacy. Ibutilide is a new intravenous therapy with considerable efficacy by with a risk of torsade de pointes. For long-term control, radiofrequency ablation is gradually supplanting chronic oral antiarrhythmic strategies.
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Dimpi Patel, Lucia Popova, Tamer S. Fahmy et al. · 2007
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Milton R. Hejtmancik, George R. Herrmann, James Y. Bradfield · American Heart Journal · 1950
Cited by 16
Adam S. Budzikowski · 2018
Cited by 1
Gregory K. Feld, Thomas McGarry · Jaypee Brothers Medical Publishers (P) Ltd. eBooks · 2015
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J P Folliot, G Ducloux, J Lekieffre · The American Journal of Cardiology · 1981
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National Cancer Institute · Definitions · 2020
A disorder characterized by an electrocardiographic finding of an organized, regular atrial rhythm with atrial rate of 240-340 beats per minute.Multiple P waves typically appear in the inferior leads in a saw tooth-like pattern between the QRS complexes.(CDISC)
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Ashok Garg, Gregory K. Feld · Current Treatment Options in Cardiovascular Medicine · 2001
Cited by 6
Melvin M. Scheinman, Yanfei Yang, Jie Cheng · Pacing and Clinical Electrophysiology · 2004
Cited by 28