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About 10,000 papers for “Atrophy”

Atrophy

Justin Rich, Tariq Walizai, Bálint Botz · Radiopaedia.org · 2020

Cited by 0Open access

Atrophy

Rok Berlot, Claudia Metzler-baddeley, Michael J O'sullivan · 2014

CSF contamination\tcontributes\tto\tapparent microstructural alterations\tin\tmild\tcognitive impairment

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Atrophy

Kluwer Academic Publishers eBooks · 2006

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atrophy

2024

Citation: 'atrophy' in the IUPAC Compendium of Chemical Terminology, 5th ed.; International Union of Pure and Applied Chemistry; 2025. Online version 5.0.0, 2025. 10.1351/goldbook.10442 • License: The IUPAC Gold Book is licensed under Creative Commons Attribution-ShareAlike CC BY-SA 4.0 International for individual terms. Requests for commercial usage of the compendium should be directed to IUPAC.

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Atrophy

SpringerReference · 2012

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Atrophy

J. H. Duffus · IUPAC Standards Online · 2016

The glossary contains definitions and explanatory notes, if needed, for over 1200 terms frequently used in the multidisciplinary field of toxicology. The glossary is compiled primarily for chemists who now find themselves working in toxicology or requiring a knowledge of the subject, especially for hazard and risk assessment Many medical terms are included because of their frequent occurrence in the toxicological literature and because chemists would not normally be expected to be familiar with them. There are two annexes, one containing a list of abbreviations used in toxicology and and one containing a list of abbreviations used by international bodies and by legislation relevant to toxicology and chemical safety.

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Atrophy

Rashid, W, Chard, DT, Miller, DH · UCL Discovery (University College London) · 2007

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Atrophy

National Cancer Institute · Definitions · 2020

Ope n Pe e r v ie w on Qe ios

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Atrophy

Early Amd · 2013

Refractive error emetropia (�0.5 to 0.5) 5163 48.3 261 39.3 154 37.1 151 39.6 63 41.2 7 19.4 3 23.1 4 17.4 hyperopia mild (�0.5 to �3) 3004 28.1 230 34.6 138 5.1 141 37.0 44 28.8 19 52.8 7 53.8 12 52.2 hyperopia mod/high (��3) 389 3.6 31 4.7 21 33.3 15 3.9 9 5.9 2 5.6 0 0.0 2 8.7 myopia mild (�0.5 to �3) 1714 16.0 120 18.1 85 20.5 59 15.5 30 19.6 6 16.7 3 23.1 3 13.0 myopia mod/high (��3) 379 3.5 15 2.3 17 4.1 10 2.6 4 2.6 0 0.0 0 0.0 0 0.0 Iris color lighter (gray/blue/green) 3715 34.7 207 31.1 135 32.4 127 33.3 57 37.3 21 58.3 10 77 11 47.8

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Atrophy

Manfred Schwab · 2016

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Atrophy

Chirag S. Desai, Rashid Shahid · 2023

Atrophy of skin, a morphological entity described under secondary skin lesions, is a result of partial or significant loss of epidermis, dermis, subcutis and/or skin appendages leading to overall thinning and loss of elasticity. Apart from primary atrophic conditions, a large number of dermatological diseases and traumatic conditions leave atrophy as sequelae.

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Spinal muscular atrophy (Werdnig‑Hoffmann atrophy disease)

Mariana A. RYZNYCHUK, Mariana I. KRYVCHANSKA, Irina V. LASTIVKA et al. · SHILAP Revista de lepidopterología · 2018

Introduction. Spinal muscular atrophy type 1 is an autosomal recessive neuromuscular disorder characterized by degeneration of the anterior horn cells in the spinal cord, leading to symmetric muscle weakness and atrophy. 95% of affected children die before 2 years of age. The annual incidence in the world has been estimated at around 1/11 000. The errors (mutations) in the SMN1 gene prevalence vary from 1: 38 to 1: 70 in the population. The disorder is primarily caused by the homozygous deletions of the gene (5q12.2-q13.3). The SMN gene mutation is primarily caused by a homozygous deletion in exons 7 or 8. Case presentations. 2 clinical cases of children with the Werdnig-Hoffmann disorder will be presented, and a literature review of this pathology. Two cases of spinal muscular atrophy diagnosed in Chernivtsi region, Ukraine, are presented. In both children, a molecular genetic analysis found the homozygous deletions of SMN1 gene in exons 7 and 8. Most affected children die within 2.3- 1.3 years of age. These two cases ended lethally due to subinfection. Material was collected in accordance with ethical standards of work person under Helsinki Declaration (World Medical Association Declaration of Helsinki, Ethical Principles for Medical Research Involving Human Subjects). Genealogical analysis of families, biochemical analysis of blood, ENMG were carried out. The molecular genetic method was used: DNA was extracted and the deletions of 7 and 8 exons of the telomeric SMN gene were tested by PCR method. The disorder usually manifests in young children, if mother has a history indicating a weak fetal movement during pregnancy. Hypotension and hypotrophy of muscles, absence of tendon reflexes on lower extremities, fibrillation of the muscles of the tongue and fingers are observed in the neonatal period. Children with this pathology can poise their heads, but never turn over and do not sit. They are characterized by a „frog“ position: the limbs are laid in the shoulder and femoral joints and bent in elbow and knee joints. Chest distortions are pathognomonic. The main cause of death is respiratory distress associated with intercurrent respiratory disorders. Conclusion. Based on the literature data and our experience of monitoring children with SMA type I, the disorder has a malignant rapidly progressing course

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Geographic atrophy

2012

CLINICAL FEATURES OF GEOGRAPHIC ATROPHY GA is easily recognized clinically, as it appears as a well-demarcated area of decreased retinal thickness, compared with the surrounding retina, with a relative change in color that allows for increased visualization of the underlying choroidal vessels. Both the location and pattern of the atrophy may vary in appearance. Drusen, usually a mixture of the soft and calcifi c types, are present in most eyes until the GA becomes so extensive as to resorb the macular drusen (2). There may be pigmentary alteration, either hypopigmentation or hyperpigmentation preceding and later surrounding the macular atrophy. Forty percent of eyes with macular GA also have peripapillary GA, which may become confl uent with the macular atrophy (7). Peripheral reticular degeneration of the pigment epithelium is present in about 41% of eyes (7). The increased choroidal vessel detail in the area of GA is usually the mosteasily identifi ed fundus change and further refl ects the extent of RPE attenuation. On fl uorescein angiography, this translates into an area of hyperfl uorescence that corresponds to the ophthalmoscopic borders of the GA, secondary to transmission defect and staining. The intensity of hyperfl uorescence from the choroidal fl ush may vary depending on the presence or absence of the underlying choriocapillaris (4). Fluorescein angiography may also aid in distinguishing GA from occult choroidal neovascularization, which may otherwise appear clinically indistinguishable. A recent paper suggests that an area that satisfi es one color photographic criterion of GA and also shows hyperfl uorescence on FA may be considered GA, allowing for earlier defi nition of this condition (11).

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[Neuromuscular atrophy].

M Krutsay · PubMed · 1995

Author reports a case of neural muscular atrophy of a 59-year-old female patient, verified by autopsy and histologic examination. The histologic picture corresponded to the both I. and III. type of sensory-motor polyneuropathyes.

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