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About 9,492 papers for “Microvessel”

Microvessel tree.

Jakob H. Lagerlöf (3352850), Peter Bernhardt (1735966) · Figshare · 2016

<p>Microvessel tree (red) inside a semi-transparent isosurface (blue), marking a partial oxygen pressure level of 0.01 mmHg.</p>

Cited by 0Open access

Microvessel density.

Louisa Bolm (3584387), Petro Zghurskyi (9115518), Hryhoriy Lapshyn (9115521) et al. · Figshare · 2020

<p>a High microvessel density, arrow pointing at densely arranged microvessels; b Low microvessel density, arrow pointing at rarely scattered microvessels. 10x magnification.</p>

Cited by 0Open access

Microvessel Growth

A. Iguchi, S. Perrey, A. M. -e. Lee et al. · 2003

Permissions: Requests for permissions to reproduce figures, tables, or portions of articles originally published in Circulation Research can be obtained via RightsLink, a service of the Copyright Clearance Center, not the Editorial Office. Once the online version of the published article for which permission is being requested is located, click Request Permissions in the middle column of the Web page under Services. Further information about this process is available in the Permissions and Rights Question and Answer document. Reprints: Information about reprints can be found online at:

Cited by 0

Quantification of microvessel density.

Yong-Guang Zhang (337213), Jing-Hui Huang (337216), Xue-Yu Hu (337219) et al. · Figshare · 2015

<p>All data are expressed as the mean ± standard error of mean. *<i>P</i><0.05 for the comparison with the L-CCH group.</p>

Cited by 0Open access

Pre-existing microvessel network.

Yan Cai (298695), Jie Wu (10038), Zhiyong Li (47765) et al. · Figshare · 2016

<p>The typical tree-like architecture network of pre-existing vessels in the basic model.</p>

Cited by 0Open access

Dexamethasone maintains microvessel integrity.

Alipio Pinto (285826), Mariana Jacobsen (435927), Patricia A. Geoghegan (285824) et al. · Figshare · 2015

<p>Fluorescence micrographs show changes in the profile of microvessels from the motor cortex after four days of treatment with saline (A and E), LPS (B and F), Stx2 (C and G) and Stx2+LPS (D and H) followed by i.p. injection of saline (A through D) or Dexamethasone (E through H). Dexamethasone rescues microvessel density (I) and glycocalyx distribution (J), similar to density and distribution in the control groups. K: negative control in mice motor cortex obtained by not adding <i>Lycopersicum</i><i>esculentum</i> lectins. *: significant differences between Dexamethasone and saline treatments.</p>

Cited by 0Open access

Freshly isolated brain microvessel structure.

Alvaro Cobo-Calvo (6788900), Anne Ruiz (479533), Chloé Richard (9338520) et al. · Figshare · 2020

<p>(a) Fresh brain microvessels are depicted after isolation, (b) pericyte staining (PDGFR- β), (c) Claudin-5, (d) Occludin, (e) PECAM, and (f) GFAP.</p> <p>(PDF)</p>

Cited by 0Open access

Regulation of brain microvessel function.

Hideyuki Kobayashi, Hiroki Yokoo, Toshihiko Yanagita et al. · Folia Pharmacologica Japonica · 2002

The brain microvessels are formed by a specialized endothelium and regulate the movement of solutes between blood and brain. The endothelial cells are sealed together by tight junctions and play a role as the blood-brain barrier. The brain microvessels express GLUT1 as the major form of glucose transporter, aquaporin-4 as a water channel, and p-glycoprotein as a xenobiotic transporter. Occludin and claudin-5 have been identified as the components of tight junction. Increasing evidence suggests that the activities of the transporters are regulated by adrenergic nerve activity as well as by bioactive peptides such as adrenomedullin. The regulation of the activity as well as expression of these transporters may become a strategy for prophylaxis and treatment of not only cerebral vascular diseases but also neurodegenerative disorders, developmental abnormalities and aging of the brain.

Cited by 0Open access

Radiation-Induced Macrovessel/Microvessel Disease

Jun‐ichi Abe, Bryan G. Allen, Andreas Beyer et al. · Arteriosclerosis Thrombosis and Vascular Biology · 2024

Radiation therapy (RT) is a cornerstone in cancer treatment (used in 50% of cases), yet challenges persist because damage to normal tissue through direct impact of radiation or bystander effects is inevitable. Injury of macrovessels by RT manifests as obstructive disease, which is akin to atherosclerotic disease. Historically observed in coronary arteries of patients treated for breast cancer and lymphoma, it also affects patients receiving contemporary therapy for lung and chest cancers. Moreover, radiation at various sites can lead to peripheral vascular disease. An aspect of radiation-induced injury that has received little attention is microvascular injury, which typically results from damage to the endothelium and is considered the primary driver of RT-induced toxicity in the skin, kidney, and brain. This review delves into the clinical manifestations of RT-induced vascular disease, signaling pathways, cellular targets affected by radiation injury, and preclinical models of RT-induced vascular injury. The goal is to inspire the development of innovative strategies to prevent RT-related cardiovascular disease.

Cited by 14Open access

Microvessel Remodeling in Cerebral Ischemia

Danica Stanimirovic, María Moreno, Arsalan S. Haqqani · Humana Press eBooks · 2008

The neurovascular (NV) unit, broadly defined as a segment of brain vasculature, is composed of functionally integrated cellular (including brain endothelial cells, astrocytes, pericytes, and smooth muscle cells) and acellular elements that form the basement membrane. Brain insults or conditions characterized by tissue hypoxia induce dramatic changes in the NV unit that include the disruption of interendothelial tight junctions, breakdown of the basal lamina, and endothelial proliferation, migration, and reorganization to form new capillaries and microvessels. This NV remodeling is controlled by a complex interplay of numerous mediators originating from cellular components of the NV unit and from breakdown of the extracellular matrix. Several of these mediator families are also involved in neuronal remodeling, thus providing for integration of vascular and neuronal responses to the ischemic state. On the global molecular scale, changes in ischemic cerebral microvessels include temporally controlled regulation of genes and proteins involved in cell cycle, blood-brain barrier phenotypic properties, cell proliferation, motility, and inflammation. The integration/coordination of angiogenic and neuronal remodeling during postischemic brain recovery is an important determinant of the overall neurological outcome.

Cited by 2

Training model for microvessel anastomosis

David C. Kim, Zahid B. M. Niazi, Peter G. Hayward et al. · Microsurgery · 1994

In the early stages of microsurgical training a model is required which is accessible, inexpensive, and approximates human tissue. In this research centre initial training exercises utilize cold stored vessels harvested from sacrificed animals used in other experiments. This model serves as an initial training tool prior to the progression to live animal research which maximizes use of animal specimens and approximates the in vivo qualities of blood vessels. The costs involved are minimal and a large number of vessels can be efficiently harvested and stored using basic equipment. As a training model, cold stored vessels have balanced cost with practicality, given an excellent approximation to living tissue and have markedly reduced our usage of live animals for teaching. This latter effect is significant at a time of increased scrutiny of the ethical use of laboratory animals.

Cited by 31

Sonographic Depiction of Microvessel Perfusion

Arthur C. Fleischer, Kenneth J. Niermann, Edwin F. Donnelly et al. · Journal of Ultrasound in Medicine · 2004

Microbubble-enhanced sonography can depict microvessel perfusion. This technique has several potential clinical applications, including assessment of tumor blood flow and changes that occur with treatment.

Cited by 37

Microvessel density in prostatic hyperplasia

S.J. Foley, David M. Bailey · British Journal of Urology · 2000

The MVD was significantly greater in the patients with haematuria, suggesting that suburothelial microvessel proliferation may play an important role in mediating haematuria associated with BPH. This is the first time that a difference has been shown at the cellular level in patients with haematuria and could form an important foundation for subsequent research.

Cited by 54

Microstreaming in a brain microvessel.

James H. Bezer (20678491), Paul Prentice (17988064), William Lim Kee Chang (20678494) et al. · Figshare · 2025

The brain slice was exposed to a 0.4-MPa<sub>pk-neg</sub>, 10-ms pulse and imaged at 5,581 fps. Streaming patterns surrounding the microbubbles were observed during sonication. The region of dye around the bubble was affected by streaming, resulting in a clearly-defined boundary. (PNG)

Cited by 0Open access